I speculated that the Growth Inhibitors reaction
was to the presence of foreign cells identified.
Many cells being identified as mutated or differing
from the accepted DNA signaling, internally, that was
the ultimate identifier, led to the overreaction of the
The affected cells in the fracture area could be a signaling
mistake, causing Growth Inhibitors to act in mass on the
area surrounding the fracture.
Infection of soft tissue stemming from the fracture area
could be a primary effect leading to infection of cell membranes?
Some damage must have been noted internally
within the cells.
Thus, triggering the Growth Inhibitor response.
Growth Inhibitors damaged cells identified, internally, altering the
DNA, to stop replication.
That was the original function of the Inhibitors.
Chemical contamination may have contributed to
Don Robbins (Growth Inhibitors Research) 2014 Feb.